AccScience Publishing / JCTR / Volume 8 / Issue 6 / DOI: 10.18053/jctres.08.202206.015
ORIGINAL ARTICLE

Bone marrow mesenchymal stem cell-derived exosomal LINC00847 inhibits the proliferation, migration, and invasion of Ewing sarcoma

Lu Huang1 Jiachao Xiong2 Jimin Fu2 Zhenhai Zhou2 Honggui Yu2 Jiang Xu2 Liang Wu2 Kai Cao2 *
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1 Department of Children Health Care, The Maternal and Children Health Hospital of Jiangxi Province, 318 Bayi Avenue, Nanchang, Jiangxi Province, 330006, China
2 The Orthopedic Hospital, The First Affiliated Hospital of Nanchang University, 1519 Dongyue Avenue, Nanchang County, Nanchang, Jiangxi Province, 330200, China
Submitted: 22 September 2022 | Revised: 7 November 2022 | Accepted: 8 November 2022 | Published: 24 November 2022
© 2022 by the Author(s). This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution -Noncommercial 4.0 International License (CC-by the license) ( https://creativecommons.org/licenses/by-nc/4.0/ )
Abstract

Background: Ewing sarcoma (ES) is one of the most lethal primary bone tumors with a poor survival rate. Current evidence suggests that extracellular vesicles (EVs) derived from bone marrow mesenchymal stem cells (BMSCs) loaded with abundant biological functional lncRNAs confer therapeutic benefits against the development of various tumors.

Aim: This study aimed to investigate the role of exosomal lncRNAs from BMSCs in the pathogenesis of ES.

Methods: Bioinformatic analysis and qRT-PCR experiments were used to detect the expression level of LINC00847 in ES tissues and cells. Cell biology experiments examined the effect of in vitro proliferation, migration, and invasion abilities and the biological function of BMSCs-derived LINC00847. Finally, we constructed a LINC00847-associated competitive endogenous RNA (ceRNA) network by in silico methods. Gene Set Enrichment Analysis (GSEA) was conducted to reveal the potential molecular mechanism of LINC00847.

Results: We found that LINC00847 was markedly downregulated in ES. Overexpression of LINC00847 inhibited ES cell proliferation, migration, and invasion. Furthermore, BMSCs-derived EVs inhibited the proliferation, migration, and invasion of ES cells by delivering LINC00847. We constructed a LINC00847 related-ceRNA network contains five miRNAs (miR-18a-5p, miR-18b-5p, miR-181a-5p, miR-181c-5p, and miR-485-3p) and four mRNAs (GFPT1, HIF1A, NEDD9, and NOTCH2).

Conclusions: Overall, this study found that BMSCs-EVs-derived exosomal LINC00847 inhibited ES cell proliferation, migration, and invasion. The ceRNA regulatory mechanism of LINC00847 may participate in the pathogenesis of the malignant phenotype of Ewing sarcoma.

Relevance for patients: These findings suggest that BMSCs-derived exosomal lncRNAs may be used for the personalized treatment of tumors, providing a novel theoretical framework for treating ES.

Keywords
Bone marrow mesenchymal stem cells
ceRNA
Ewing sarcoma
Extracellular vesicles
LINC00847
Conflict of interest
The authors declared no conflicts of interest.
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Journal of Clinical and Translational Research, Electronic ISSN: 2424-810X Print ISSN: 2382-6533, Published by AccScience Publishing